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"A single arginine to tryptophan interchange at beta-chain residue 458 of human complement component C4 accounts for the defect in classical pathway C5 convertase activity of allotype C4A6. Implications for the location of a C5 binding site in C4.." The Journal of Immunology
148.9
(1992):
2803-2811.
Web. 25 Jan. 2021.