Mechanism of Prostaglandin (PG)E2-Induced Prolactin Expression in Human T Cells: Cooperation of Two PGE2 Receptor Subtypes, E-Prostanoid (EP) 3 and EP4, Via Calcium- and Cyclic Adenosine 5′-Monophosphate-Mediated Signaling Pathways

Sarah Gerlo; Peggy Verdood; Birgit Gellersen; Elisabeth L. Hooghe-Peters; Ron Kooijman

doi:10.4049/jimmunol.173.10.5952

Mechanism of Prostaglandin (PG)E₂-Induced Prolactin Expression in Human T Cells: Cooperation of Two PGE₂ Receptor Subtypes, E-Prostanoid (EP) 3 and EP4, Via Calcium- and Cyclic Adenosine 5′-Monophosphate-Mediated Signaling Pathways

Sarah Gerlo, Peggy Verdood, Birgit Gellersen, Elisabeth L. Hooghe-Peters and Ron Kooijman

J Immunol November 15, 2004, 173 (10) 5952-5962; DOI: https://doi.org/10.4049/jimmunol.173.10.5952

Sarah Gerlo

*Laboratory of Neuroendocrine Immunology, Department of Pharmacology, Free University of Brussels, Brussels, Belgium; and

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Peggy Verdood

*Laboratory of Neuroendocrine Immunology, Department of Pharmacology, Free University of Brussels, Brussels, Belgium; and

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Birgit Gellersen

†Endokrinologikum Hamburg, Hamburg, Germany

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Elisabeth L. Hooghe-Peters

*Laboratory of Neuroendocrine Immunology, Department of Pharmacology, Free University of Brussels, Brussels, Belgium; and

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Ron Kooijman

*Laboratory of Neuroendocrine Immunology, Department of Pharmacology, Free University of Brussels, Brussels, Belgium; and

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The Journal of Immunology

Vol. 173, Issue 10

15 Nov 2004

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Mechanism of Prostaglandin (PG)E₂-Induced Prolactin Expression in Human T Cells: Cooperation of Two PGE₂ Receptor Subtypes, E-Prostanoid (EP) 3 and EP4, Via Calcium- and Cyclic Adenosine 5′-Monophosphate-Mediated Signaling Pathways