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Table of Contents

February 1, 1916; Volume 1,Issue 1
  • You have accessRestricted access
    Studies in Anaphylaxis
    Richard Weil
    J Immunol February 1, 1916, 1 (1) 1-18;
  • You have accessRestricted access
    Studies in Anaphylaxis
    Richard Weil
    J Immunol February 1, 1916, 1 (1) 19-34;
  • You have accessRestricted access
    Studies in Anaphylaxis
    Richard Weil
    J Immunol February 1, 1916, 1 (1) 35-46;
  • You have accessRestricted access
    Studies in Anaphylaxis
    Richard Weil
    J Immunol February 1, 1916, 1 (1) 47-49;
  • You have accessRestricted access
    Complement Fixation in Varicella
    John A. Kolmer
    J Immunol February 1, 1916, 1 (1) 51-58;
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    Complement Fixation in Vaccinia and Variola
    John A. Kolmer
    J Immunol February 1, 1916, 1 (1) 59-81;
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    The Fate of Various Antibodies in the Precipitin Reaction
    Frederick P. Gay and Ruth L. Stone
    J Immunol February 1, 1916, 1 (1) 83-104;
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    Kidney Lesions in Chronic Anaphylaxis
    T. Harris Boughton
    J Immunol February 1, 1916, 1 (1) 105-118;
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    The Site and Rate of Destruction of Pneumococci Following Intraperitoneal Injection
    F. Berry and C. O. Melick
    J Immunol February 1, 1916, 1 (1) 119-124;
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    Scientific Proceedings of the Society for Serology and Hematology New York
    J Immunol February 1, 1916, 1 (1) 125-128;
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The Journal of Immunology
Vol. 1, Issue 1
1 Feb 1916
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Immunodominant AH1 Antigen-Deficient Necroptotic, but Not Apoptotic, Murine Cancer Cells Induce Antitumor Protection
Osteoclasts Derive Predominantly from Bone Marrow–Resident CX3CR1+ Precursor Cells in Homeostasis, whereas Circulating CX3CR1+ Cells Contribute to Osteoclast Development during Fracture Repair
Intracellular IL-1 Receptor Antagonist Isoform 1 Released from Keratinocytes upon Cell Death Acts as an Inhibitor for the Alarmin IL-1α
Polysaccharide Capsules Equip the Human Symbiont Bacteroides thetaiotaomicron to Modulate Immune Responses to a Dominant Antigen in the Intestine
Natural and Induced Tolerogenic Dendritic Cells
Cutting Edge: IL-6–Driven Immune Dysregulation Is Strictly Dependent on IL-6R α-Chain Expression

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Print ISSN 0022-1767        Online ISSN 1550-6606