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A Toxic Ocular Reaction

II. On the Nature of the Reaction

From the Laboratories of The Mount Sinai Hospital, New York City, and the Department of Ophthalmology of Columbia University, College of Physicians and Surgeons, N. Y. C.

Abstract

The work embodied in this paper demonstrates that increased permeability of the ciliary body is a major factor in the mechanism of the toxic ocular reaction and that the fibrinogen system plays an essential role in coagulation of the plasmoid-toxic aqueous. It also provides evidence for the toxic nature of the reaction and for the local influence of the toxic ocular principles upon the ocular vessels. The histologic study of the eyes removed during the reaction shows a moderate number of polymorphonuclear leucocytes peri- and paravascularly in the uvea and in the retina. A retroocular injection of adrenaline (Ayo-Herrmann experiment) protects the eye against the toxic ocular reaction.

Footnotes

¹ Knapp Fellow for 1941–1942 in Columbia University, College of Physicians and Surgeons, N. Y. C.; formerly Moritz Rosenthal and Henry F. Dazian Fellow in The Mount Sinai Hospital, N. Y. C.