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The Complement Profile in Babesiosis¹

Department of Pathology, University of Connecticut Health Center, Farmington, Connecticut 06032

Abstract

Rats have been infected with Babesia rodhaini, a protozoan agent that induces immune complex disease. Assays for complement have revealed depletion of C2, C3, C4, C5, and whole complement in the course of infection. No evidence of depletion in the alternative (properdin) pathway was found. These findings are consistent with the conclusion that the classical complement pathway is activated during the course of the protozoan infection.

Footnotes

¹ This work was supported in part by National Institutes of Health Grant AI-09651.